The following clinical encounters deserve attention, and will be amplified in the ensuing discussion.
- Aspartame reactors being treated with insulin or an oral drug sometimes could not decide if they were experiencing a reaction to insulin or to aspartame.
- Several insulin-dependent diabetics seemed more prone to aspartame-induced convulsions- both grand mal (see Case III-24) and petit mal (see Case III-25).
- Insulin resistance and diabetic control tended to improve promptly after avoiding aspartame products.
- Questions arose about missed or doubled dosages of insulin or oral drugs due to aspartame-induced confusion (Chapter VI).
- A few of my diabetic patients (not included in this series) had abstained from aspartame before I queried them about its use because they already had concluded that it aggravated their condition.
- Aspartame addiction (Chapter VII - G) compounded the problem when diabetics used these products to get their "sugar fix."
A. PRECIPITATION OF CLINICAL DIABETES
Several patients whom the author had followed for years evidenced a prior diabetic response ("decreased glucose tolerance") by glucose-tolerance testing - that is, their blood-glucose concentrations after drinking a glucose load exceeded 200 mg percent. On an appropriate diet, the fasting blood sugar (FBS) concentrations remained normal (115 mg percent or less) or only slightly elevated. Their subsequent elevated values (hyperglycemia), along with weakness and other symptoms of diabetes, proved puzzling until it was ascertained they used aspartame products. Clinical and biochemical improvements occurred when such products were stopped.
In addition, some patients with longstanding reactive hypoglycemia were founded to have fasting hyperglycemia for the first time after consuming aspartame (see Case XIII-2 and XIII-3). Their FBS values then normalized after abstinence.
Three aspartame reactors developed diabetes after attacks of apparent aspartame induced pancreatitis (Chapter IX-D). Case XIII-4 illustrates this sequence.
Representative Case Reports
Case XIII-1
A 47-year-old man with hemochromatosis or iron storage disease had been attended several years. (This condition is also known as "bronze diabetes" because iron deposits in the pancreas can damage the islet cells that produce insulin.) When seen in July 1986, his FBS had risen to 159 mg percent. On direct inquiry, he stated that he had been drinking considerable aspartame sodas. The FBS declined to 97 mg percent three weeks after abstinence.
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